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Dexketoprofen is the (S)-enantiomer, which is believed to confer analgesia. Theoretically dexketoprofen is expected to provide equivalent analgesia to ketoprofen at half the dose, with a consequent reduction in gastrointestinal adverse events. This review is one of a series on oral analgesics for acute postoperative pain.
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Dexketoprofen, an NSAID used in the management of acute and chronic pains, is licensed in several countries but has not previously been the subjected of a systematic review. We used published and unpublished information from randomised clinical trials (RCTs) of dexketoprofen in painful conditions to assess evidence on efficacy and harm. Methods
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Dexketoprofen supplementation benefits to improve pain control at 48 hours and reduce the need for rescue drug in migraine attack patients. Keywords: dexketoprofen, migraine attack, pain control, randomized controlled trials Go to: 1. Introduction
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Description. Dexketoprofen is a monocarboxylic acid that is (S)-hydratropic acid substituted at position 3 on the phenyl ring by a benzoyl group. A cyclooxygenase inhibitor, it is used to relieve short-term pain, such as muscular pain, dental pain and dysmenorrhoea. It has a role as a non-steroidal anti-inflammatory drug, a cyclooxygenase 1.
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Dexketoprofen is the (S)โenantiomer, which is believed to confer analgesia. Theoretically dexketoprofen is expected to provide equivalent analgesia to ketoprofen at half the dose, with a consequent reduction in gastrointestinal adverse events. This review is one of a series on oral analgesics for acute postoperative pain.
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Objective: Dexketoprofen trometamol is a modified non-selective COX inhibitor with a rapid onset of action that is available as both oral and parenteral formulations. The aim of this narrative review was to assess the efficacy and tolerability/safety of dexketoprofen trometamol in acute pain states using the best available published scientific evidence (randomized controlled clinical trials.
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Dexketoprofen is a nonsteroidal anti-inflammatory drug (NSAID). It is manufactured by Menarini, under the tradename Keral. It is available in the UK, as dexketoprofen trometamol, as a prescription-only drug and in Latin America as Enantyum, produced by Menarini.
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Dexketoprofen. In Meyler's Side Effects of Drugs (Sixteenth Edition), 2016. Drug-drug interactions Co-magaldrox. The effect of co-magaldrox (magnesium trisilicate + aluminium hydroxide) on the pharmacokinetics of dexketoprofen trometamol has been studied in a randomized, three-way, crossover study in 24 healthy volunteers who took three single doses of dexketoprofen trometamol 25 mg either.
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1. Introduction. Dexketoprofen has analgesic, anti-inflammatory, and antipyretic properties, and belongs to non-steroidal anti-inflammatory drugs [1].Dexketoprofen is a propionic acid derivative - (S+) enantiomer of ketoprofen [1], [2].The action of the drug is mainly based on the inhibition of cyclooxygenases: constitutive (cyclooxygenase-1) responsible for the synthesis of prostaglandins.
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Authors' conclusions. Ketoprofen at doses of 25 mg to 100 mg is an effective analgesic in moderate to severe acute postoperative pain with an NNT for at least 50% pain relief of 3.3 with a 50 mg dose. This is similar to that of commonly used NSAIDs such as ibuprofen (NNT 2.5 for 400 mg dose) and diclofenac (NNT 2.7 at 50 mg dose).
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Dexketoprofen is an enantiomer of ketoprofen (S+) that belongs to nonsteroidal anti-inflammatory drugs and has analgesic, anti-inflammatory, and antipyretic properties. Dexketoprofen has a stronger effect than ketoprofen, which makes it a readily used preparation. The review aims to find in recent o.
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Tramadol/dexketoprofen is a new pharmacological association with analgesic activity. It is a combination of dexketoprofen, a NSAID with both peripheral and central antinflammatory activity, and tramadol, a weak opioid with double mechanism of action, partial agonism on the ฮผ-opioid receptor and inhibition of monoamines (serotonine and.
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ketoprofen and sparsentan both increase nephrotoxicity and/or ototoxicity. Use Caution/Monitor. Coadministration of NSAIDS, including selective COX-2 inhibitors, may result in deterioration of kidney function (eg, possible kidney failure). Monitor for signs of worsening renal function with concomitant use with NSAIDs.
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Hypersensitivity to dexketoprofen, or any other NSAIDs. History of asthma attacks, bronchospasm, angioneurotic oedema, acute rhinitis, nasal polyps, urticaria, or other allergic-type reaction after taking aspirin or other NSAIDs; known photoallergic or phototoxic reactions during therapy with ketoprofen or fibrates; active or suspected peptic ulcer or haemorrhage, history of gastrointestinal.
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In addition, dexketoprofen supplementation was associated with a significantly decreased need for the use of rescue medication (RR, 0.64; 95% CI, 0.43-0.94;.
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